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Colchicine decreases leukocyte chemotaxis {04} {06} and phagocytosis {01} {02} {03} {04} and inhibits the formation {04} {06} {08} and release {04} {06} {08} {09} of a chemotactic glycoprotein that is produced during phagocytosis of urate crystals {04} {06} {08}. However, colchicine does not reduce the frequency or severity of ocular lesions associated with this disease or improve visual acuity in affected patients {22} {23} {25}. Colchicine may also increase degradation of collagen {29}by stimulating production of collagenase {42}. Although colchicine may retard the development of fibrosis and hepatic failure in patients with biliary cirrhosis {32} {34}, it does not relieve symptoms {04} {26} {28} {31}, prevent or reverse histological changes characteristic of the disease {04} {26} {28} {29} {31}, or decrease the need for hepatic transplantation {32}. Also, colchicine corrects some of the abnormalities of lymphocyte and monocyte function that have been identified in patients with active biliary cirrhosis {34}. Colchicine also inhibits urate crystal deposition, which is enhanced by a low pH in the tissues, probably by inhibiting oxidation of glucose and subsequent lactic acid production in leukocytes {01} {02} {03}.

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